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Myotonia

One of my relations has been diagonised as suffering from Myotonia. Please enlighten me about the disease and the remedy.
 
  maya_hari on 2005-03-01
This is just a forum. Assume posts are not from medical professionals.
Its a nervous disorder somewhat as below.
As AMP_ADP_ATP could be involved some of the Phos remedies may be of use but this would need a detailed cover of all the characteristics of the person involved.

Be careful and select a 1st class homeopath.



When a nerve impulse arrives in the presynaptic terminal it opens voltage-gated Ca2+ channels, producing a rise in the intracellular Ca2+ concentration ( [Ca2+]i) that triggers the exocytosis of synaptic vesicles. The amount of transmitter released varies with [Ca2+]i and thus with the magnitude of the presynaptic Ca2+ current. In turn, this is influenced by the duration of the membrane depolarization and thus by the amplitude of the voltage-gated K+ current that underlies membrane repolarization. A reduction in the presynaptic K+ current therefore leads to excess transmitter release and postsynaptic hyperexcitability, as in episodic ataxia type 1 and acquired neuromyotonia. Conversely, a reduction in the presynaptic Ca2+ current is associated with reduced transmitter release, as occurs in the Lambert–Eaton myasthenic syndrome when the density of presynaptic Ca2+ channels is decreased by receptor internalization induced by the binding of autoantibodies.
Once released, the transmitter diffuses across the synaptic cleft and binds to receptors in the postsynaptic membrane. At the neuromuscular junction, for example, acetylcholine (ACh) binds to the nicotinic acetylcholine receptor (AChR), and opens an intrinsic ion channel. The resulting synaptic current produces a depolarization of the postsynaptic membrane (the endplate potential) which, if it is sufficiently large, triggers an action potential in the muscle fibre. A reduction in AChR density, as in myasthenia gravis, decreases effective transmission and leads to muscle weakness. Gain-of-function mutations in AChR may also induce myasthenia, by causing prolonged depolarization of the postsynaptic membrane and thereby Na+ channel inactivation. This depolarizing block is the basis of the slow-channel syndromes. Mutations in the voltage-gated Na+ channel of skeletal muscle may cause paralysis, or myotonia.
 
passkey last decade
My relative suffering from Myotonia also has Sclerosis which is progressive. His hands and legs shake a lot. His neck falls down and he must conciously keep it up..
He is on allopathy treatment and is taking the following medicines: Endoxan 500 mg for the sclerosis, Medrol 4mg once, Trental 400 twice, Ecosprin 75 mg once, Eltroxin 100 mg once and Mexitol (for the Myotonia) thrice. He is also under homeopathy who has given him Gelsemium200, Arnica 10M and Ichthyolium 30 on 15h Jan and then on 22nd Feb Agaricus 200, 1M, Calc Phos 12x and Hypericum 30. I dont know the frequencies.
Yes Passkey, (walkin) he is under treatment by one of the best homeopaths in Calcutta, India. But after hearing about the forum, he has requested if anyone here can help him.
 
maya_hari last decade
Dear Maya hari,
for maytonia very few medicine is in homoeopathy. for cure of your disease counsiling is required to know the detail. for that you have to send all the detail by email. with photographs of the finger.
dr.d.sharma
 
deoshlok last decade

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