Homeopathy and Health Forum
Gilbert's syndromeI have been diagonised with gilberts syndrome...bilirubin between 1.5 and 2.0. Other symptoms include fatigue, low appetite and stomach upset. Can this be cured permanently?
vikram_bansal1978 on 2006-11-10
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Immidiately start Natrum Sulphuricum 3x or 6x thrice a day.
Chilidonium 6c twice a day.
Carduus Mariuns Q ten drops thrice a day.
kuldeep on 2006-11-10and be ready to go to ER, in case some thing goes wrong.
Please read the thread
Dr.luc's article on pseudo homeopathy.
it is floating around on the main page of the forum.
gavinimurthy on 2006-11-10Gilberts Syndrome is hard to cure since it a congenital problem.
But from Kuldeep's prescription u will get immense relief and u won't have to go to ER.
I prefer Chelidonium and Cardus Marianus both in mother tincture form. But u can also use as Kuldeep has advised.
Chelidonium acts on Rt lobe of liver and Cardus Marianus has afinity for Lt Lobe of liver, both are required for your treatment. In Gilbert's Syndrome whole liver is affected not just any one lobe.
You should also take plenty of fluids like glucose and avoid junk foods and drinks (both soft and hard)throughout your life.
drprodip on 2006-11-12This thread continues beneath the following ad.this is what Clarke says in his discussion on Cardus Marianus.
'The liver enlargement of Cardus M is in the transverse direction.(That of chel. being more vertical)'
gavinimurthy on 2006-11-12
GILBERT'S SYNDROME - a patient's guide
Dr Philip Wong - Gastroenterologist and Hepatologist
Gilbert's syndrome is a common inherited disorder that affects the processing by the liver of the pigments in the bile called bilirubin. This results in an increase in the level of bilirubin levels in the bloodstream and can lead to a yellowing of the skin and the eyes, that is jaundice. The liver itself is otherwise normal and people with Gilbert's syndrome have a normal life expectancy. It is common and not a dangerous condition in any way.
Gilbert's syndrome is arguably the most common medical syndrome identified. In normal people bilirubin is bound (conjugated) to specific protein(s) by a liver enzyme, called bilirubin-uridine glucuronosyltransferase (bilirubin-UGT), which allows it to be excreted in the bile. It has long been recognised that bilirubin-UGT activity measured in liver biopsies of individuals with Gilbert's syndrome is almost invariably reduced, suggesting that reduced enzyme activity is the source of the impairment in bilirubin metabolism. Thus in Gilbert's syndrome bilirubin is less efficiently excreted into the bile leading to elevated levels of unconjugated bilirubin in the bloodstream.
Clinical and laboratory findings
Gilbert's syndrome is most often recognised in the second or third decade of life and is rarely diagnosed before puberty. The diagnosis is made most often following routine screening blood tests or when fasting associated with surgery or intercurrent illness unmasks the hyperbilirubinemia. Gilbert's syndrome affects 3 to 7% of the population, with males predominating over females by a ratio of 2-7:1.
The normal bilirubin level in the blood is less than 20 umol/L while in Gilbert's syndrome the bilirubin level is usually mildly raised. In most cases is less than 50 umol/L and is always less than 100 umol/L. There is considerable daily and seasonal fluctuation. The bilirubin may be normal on occasion in up to one-third of patients.
Because the liver is otherwise normal in individuals with Gilbert's syndrome, physical examination is normal apart from the occasional presence of jaundice. At least 30% of patients with Gilbert's syndrome are asymptomatic, apart from the presence of mild jaundice, and are unaware of the abnormality until it is detected by incidental laboratory examination or in the course of family studies. Some people may experience a variety of nonspecific and varied symptoms, including vague abdominal discomfort, fatigue, or malaise In general, these symptoms do not correlate with the plasma bilirubin level and are more likely related to anxiety rather than to the underlying disorder in metabolism. In a third group of patients, hyperbilirubinemia may be unmasked during fasting, which usually produces an increase in the plasma unconjugated bilirubin level. Thus, sub clinical individuals may first be detected in association with caloric withdrawal due to an intercurrent febrile illness, 'morning sickness' of pregnancy or postoperatively.
Diagnosis and Treatment
The diagnosis of Gilbert's syndrome is suggested by the clinical finding of mild chronic unconjugated hyperbilirubinemia. A family history should be sought and clinical evidence of other liver or blood disorders, including haemolysis (abnormal breakdown of red cells) excluded. Further confirmation of the diagnosis may be achieved by determining the effect of fasting or intravenous nicotinic acid administration on the blood bilirubin concentration. Either manipulation generally results in a two- to threefold rise in plasma bilirubin level, even if the initial bilirubin concentrations are normal. However, not all patients with Gilbert's syndrome respond to these tests, which appear less sensitive and specific for females than males. Other diagnostic manoeuvres that have been advocated are measurement of the effect of phenobarbital on plasma bilirubin concentration, and the administration of a tracer dose of radiolabeled bilirubin for estimation of the percentage of the dose remaining in the plasma after 4 hours.
Given the above considerations, a presumptive diagnosis of Gilbert's syndrome can be made in an essentially asymptomatic individual if he /she has (1) unconjugated hyperbilirubinemia on several occasions; (2) normal results of a complete blood cell count, blood smear, and reticulocyte count; and (3) normal liver enzyme tests (see article on liver function tests) and (4) other disease processes have been excluded. If no further laboratory abnormalities develop on two or three follow-up tests during the next 12 to 18 months, then the presumptive diagnosis becomes definitive. It is conceivable that genetic testing may play a role in the diagnosis of this condition in the future.
Once the diagnosis is established, the most important aspect of management is to reassure the patient with regard to the benign and inconsequential nature of the disorder and the excellent prognosis and to prevent further unnecessary investigations.
Source : http://www.medic8.com/healthguide/articles/gilbertsyn.html
gavinimurthy on 2006-11-12In Gilbert's syndrome, the liver is normal.
The rise in the bilirubin is an enzyme defect.
Why are we assuming that, the liver is effected, and prescribing one medicine for left side, and another for right side?
In all probability this prescription may make the liver enlarged,as prolonged dosage, may induce proving.
The best thing for this patient is to reassure him that, there is nothing wrong with him, as suggested in the above article.
gavinimurthy on 2006-11-12Murthy,
You are quoting from a website of allopathic doctors.
Homeopathy has answers for his fatigue, low appetite and stomach upset.
Pl. post your full case.
Illnesses from child hood till now.
What are the other symptoms
that you are experiencing at this stage.
PANKAJ VARMA on 2006-11-12This thread continues beneath the following ad.The prescriptions are based on allopathic diagnosis.
I am questioning the very basis of these prescriptions.
If you want to treat him based on signs and symptoms, you are most welcome.
But, please don't try to defend the undefendable.
The patient would have landed into problems, had he followed those prescriptions.
gavinimurthy on 2006-11-12
Nope! It can't be "cured". I have GS also and it is totally a management game. Ive had it for about 14 years. It s not curable but I have learned it is manageable. After TONS of research, I do two things... one, stay hydrated and manage sleep. and two, I take 3 different supplements, one of which is Taurine its changed my life! The resource I use is GSLiverHealth . com. It s a website for those with Gilbert's Syndrome. I hope this helps you! Good luck! David
nddave on 2018-01-11Thanks for posting nddave. I have a relative with this-
And some people notice nothing and others
Have to really watch what they eat and drink as
It upsets digestion and seems to be trial and error process
Until one figured out what works for them.
Will send them the website. Also thanks to Murthy
For posting common sense on this thread.
simone717 on 2018-01-11
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